Lutein Levels Linked To A More Agile And “Youthful” Brain

Lutein and CognitionA new study finds a high intake of Lutein may slow down cognitive decline.

60 healthy adults between the ages of 25 and 45 participated in this study.  Carotenoid levels were assessed by measuring MPOD (Macular Pigment Optical Density), which is considered a trusted indicator of Lutein levels in the brain. Event related brain activity was used as a gauge of cognitive function and was recorded as the participants performed cognitive control testing.  Study results showed MPOD was related to both specific electrical brain activity and age during the decision making process, known as the P3 wave.  Younger adults showed a larger abundance of P3 than the older participants however, the older participants with higher MPOD levels displayed P3 measures equal to the younger participants.  Researchers concluded that Lutein appears to have a protective role in the brain since the study data indicated that participants with more Lutein were able to utilize more cognitive resources to finish their tasks.

Besides being beneficial to brain health, Lutein and Zeaxanthin have been shown to provide support for vision and vision diseases since Lutein appears to accumulate in both the brain and in the eyes.  The ability of Lutein in brain processing, memory, speed and processing is intriguing since Lutein cannot be manufactured on its own in the body.

Further study is warranted.

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Folic Acid and Dementia: Supplementation Benefits Elderly People with Mild Cognitive Decline

Folic Acid and Cognitive ImpairmentElderly people with mild cognitive impairment saw significant improvements in both cognitive performance and reduced inflammation when supplementing with 400 mcg of Folic Acid daily for a period of 12 months.

Over 150 seniors with mild cognitive impairment were randomly assigned to two groups.  One group received daily Folic Acid supplementation (400 mcg) and the other group was a conventional-treatment group.

Significant improvements in cognitive function were seen in the group supplementing with Folic Acid.  Additionally a significant reduction in levels of inflammatory cytokines was seen.  Peripheral inflammatory cytokines appear to be biomarkers for identifying individuals who may be at a greater risk of developing Alzheimer’s disease or dementia. Researchers believe looking at the role of inflammatory markers at the onset of dementia, before full clinical dementia syndrome has developed, is essential.  Researchers concluded that folic acid has significant memory enhancing and anti-inflammatory properties.

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Omega-3 Could Aid Alzheimer’s Prevention

Omega-3 and Alzheimer's DiseaseNew research finds individuals with high Omega-3 intakes have an increase in blood flow in the brain suggesting a link might exist between Omega-3 and Alzheimer’s Disease.

Over 160 randomly selected people participated in this trial.  These participants were taken from clinics and their brain function was studied using single photon emission computed tomography (SPECT).  SPECT measures blood perfusion in the brain.  128 regions of the brain were used to collect data on the correlation between Omega-3 blood levels as well as blood flow in the brain.  Resting-state scans were performed while participants were sitting in a dimly lit room with ambient noise and with their eyes open.  For on-task scans participants completed Continuous Performance Tests (CPTs) while the scans were performed.

Significant statistical relationships were found between EPA and DHA (Omega-3 Essential Fatty Acids)  levels in the blood and cerebral perfusion in various regions of the brain: areas known for memory encoding and retrieval (parahippocampal gyrus), areas for episodic memory, visuospatial processing, and aspects of consciousness (the right preuneus), and areas associated with bodily locomotion and posture (vermis subregion).

Researchers concluded that Omega-3 Essential Fatty Acids (EPA and DHA) may improve brain physiology which in turn leads to better cognitive reserves.

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Vitamin A Status At Birth Linked to Long-Term Risk of Cognitive Impairment and Alzheimer’s

Cognitive FunctionA new study has found elderly individuals with low Vitamin A levels may be more at risk of developing Alzheimer’s disease along with lower brain functioning.  Additionally the study found the even a marginal Vitamin A deficiency at birth might affect long-term risk factors.

This new study was made up of findings from mouse models as well as human population data.  The population study revealed that 75% of those with either a significant or even mild Vitamin A deficiency had some cognitive impairment compared to 47% with normal Vitamin A levels that experienced cognitive impairment.  The research on mice confirmed this finding.  Even a mild Vitamin A deficiency during pregnancy or immediately after birth was associated with an increase in the production of amyloid-beta plaques which are associated with Alzheimer’s disease.  Researchers found that mice deprived of Vitamin A performed poorly on standardized tests of memory as well as learning as adults.  Mice that had Vitamin A withheld while they were in the womb but were given a normal diet after they were born had lower performance in general when compared to mice receiving adequate Vitamin A in the womb but were deprived Vitamin A after birth.  It was found that some reversal of decline could be reversed with adequate Vitamin A supplementation.  Researchers concluded that monitoring Vitamin A during infancy as well as during pregnancy and eliminating a prenatal Vitamin A deficiency may aid in halting Alzheimer’s disease development.

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